Dawn's picture

This two-lead rhythm strip clearly shows the transition from normal sinus rhythm to a paroxysmal supraventricular rhythm.  In this case, the arrhythmia is AV nodal reentrant tachycardia, AVNRT.  The rate of the first rhythm, NSR, is around 75 per minute.  The fourth beat on the strip is a PAC which initiates the paroxysm of tachycardia lasting 12 beats.  The arrhythmia terminates spontaneously at that point.  The tachycardia rate is about 150/min.

The topic of supraventricular tachycardias can be a very complex one to teach.  For an excellent example of a concise lesson geared toward Primary Practice physicians, go to Dr. Grauer's VIDEO - Part III of his Arrhythmia series.

To cover the important points for the beginner-level student:

  *  It can be difficult to determine a rhythm is SVT if the rhythm is near 150 bpm and you DON'T see the beginning or end of the arrhythmia.  If the onset (or offset) is sudden, then this is not a sinus rhythm.  The sinus node speeds and slows more gradually - it doesn't change rates in one heartbeat.  This strip has an excellent view of BOTH the onset and the offset.

  *  The faster the rate, the more likely we are looking at a PSVT rather than sinus rhythm.  If a sinus tachycardia exists, we can almost ALWAYS see the reason for it in the patient's clinical situation.  We may see fever, dehydration, bleeding, fear, pain, exercise.  Therefore, a patient at rest with a rate of 150 would be suspect for PSVT.  A patient on a treadmill for 5 minutes would be considered to have a sinus rhythm.

  *  Any patient with a rate around 150 per minute should be evaluated for ATRIAL FLUTTER with 2:1 conduction.  Atrial flutter often conducts at that ratio, because a rate of 150 is fairly easy for the AV node to conduct, whereas the instrinsic rate of atrial flutter (250-350) is not.  A 12-lead ECG makes it easier to search for tell-tale flutter waves.

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ekgpress@mac.com's picture

     As per Dawn - the ECG seen here illustrated AVNRT ( = AV Nodal Reentry Tachycardia). Definitive diagnosis can be made because both onset and offset of the rhythm has been captured. I'll briefly expand on Dawn's comments.
  • It is indeed good that onset and offset of the rhythm has been captured - since the QRS complex in this unmarked lead is wide! It clearly measures 0.11 second - or more than half a box in duration. Although most VT is wider than this - it is possible to have fascicular VT with a QRS of 0.11 second. If all we had was the run (without seeing onset and offset of this rhythm) - then we would have a regular WCT (Wide-Complex Tachycardia) - and could not be certain that this rhythm was supraventricular. Seeing identical QRS morphology during the run as before and after (during sinus rhythm) confirms supraventricular etiology.
  • There is much "noise" on this tracing. This results in underlying baseline undulation that makes it impossible to tell if there is ongoing retrograde conduction during the tachycardia. Ideally one would document the rhythm with a 12-lead during tachycardia - but given the degree of artifact, it is unlikely that retrograde activity will be seen.
  • That said, despite the artifact - extra peaking of the T wave of beat #3 IS seen. This highlights occurrence of the "tell-tale" PAC which so often heralds the onset of AVNRT. Early occurrence of a supraventricular beat finds the alternate (usually slow) AV nodal pathway receptive to conduction - which if it occurs at precisely the "right moment" - catches that narrow window of time in which the fast pathway is able to conduct retrograde - and the cycle begins. Spontaneous termination of AVNRT often results from the same phenomenon - namely, a PAC that blocks one of the nodal pathways, thereby interrupting the cycle. Excessive artifact here makes it impossible to tell if this is the reason this AVNRT stops.
For those with interest in an example of identifying retrograde atrial activity during AVNRT - You may be interested in this pdf on Section 14.3 (excerpted from my ACLS-2013-ePub). Begin reading the "Beyond-the-Core" Section (14.3.7) for detailed illustration. The video link Dawn provides should consolidate key concepts.
 

Ken Grauer, MD  www.kg-ekgpress.com   [email protected] 

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