This rhythm strip was obtained from a man who was suffering an acute inferior wall M.I.  There are ST elevation and hyperacute T waves.  The rhythm is SINUS ARRHYTHMIA WITH SECOND-DEGREE AV BLOCK, TYPE II.    There is also first-degree AV block.

There are more P waves than QRS complexes, with a 3:2 ratio.  The atrial rate varies between 55 -68 beats per minute.  The sinus rate speeds slightly after the dropped QRS in each group. The ventricular rate is about 40 bpm, with grouped beating. (Regularly irregular.)

The PR intervals are steady at 226 ms (slightly prolonged).

Second-degree AV block can either be Type I (Wenckebach) or Type II.  In either case, some P waves are conducted to the ventricles, and some are not. Type I blocks usually occur in the AV node, and are often benign. Type II blocks are more often "sub-Hisian", or fascicular blocks, and are more likely to progress to higher levels of AV block and bradycardia.  When a second-degree AVB is conducted in a 2:1 ratio, it is difficult to differentiate Type I from Type II.  Features that favor the diagnosis of Type I are narrow QRS complex and the non-conducted P waves land on the previous T waves - during the refractory period of the ventricles.

Type II blocks are more likely to have a wide QRS with a bundle branch block morphology.  That is because Type II blocks often reflect serious fascicular disease.  A typical Type II block is a persistent bifascicular block (ex: RBBB and left anterior hemiblock)) with an intermittent block in the third fascicle.  Another way to think of it is an intermittent tri-fascicular block. If that one remaining fascicle stops conducting, the patient will be in complete heart block.

Signs of Type II blocks include the wide QRS and also two or more non-conducted P waves in a row.  Also, P waves that are "out in the open", away from the refractory period, but fail to conduct are an ominous sign.

One strategy for reacting to a 2:1 block is to first assess the ventricular rate (54 bpm in this example).  Determine if it is adequate for the patient's hemodynamic stability.  If not, act to increase the rate.  Otherwise, it may be prudent in the stable patient to watch the rhythm strips for a while.  Sometimes, two p waves in a row will conduct - unmasking either progressive prolongation of the PR interval (Type I) or stable PR intervals (Type II). 

The patient in this example was having an inferior wall M.I.  The ST elevation will not always show up on a monitor strip, as it does here.  A 12-lead ECG is the minimum standard for evaluating for coronary artery disease and acute M.I.  It is possible that the 2:1 block will disappear when the atrial rate (about 108 here) is slowed.

This ECG is taken from an elderly woman who complains of feeling weak and tired. We have no other clinical information, unfortunately.

There is an obvious bradycardia, with more P waves than QRS complexes.  Here is what we see:

*  Atrial rate is around 115/min. and P waves are regular and all alike.

*  Ventricular rate is around 35/min. and QRS complexes are regular and all alike.

*  PR intervals, when they occur, are all the same at 162 ms.

*  QRS duration is wide at 122 ms.

*  QTc interval is prolonged at 549 ms.

What does this mean?  There is sinus tachycardia with second-degree AV block because the atrial rate is over 100/min, but not all P waves are conducted.  The AV block looks like a Type II (Mobitz II) block because the PR intervals are all the same.  This is a reliable indicator of conduction. (Not third-degree AVB).  The wide QRS complexes are due to right bundle branch block.  The ECG signs of RBBB are: 1) wide QRS; 2) supraventricular rhythm; and 3) rSR’ pattern in V1 and Rs, with a wide little s wave, in Leads I and V6.

This ECG is from a 78-year-old woman.  We do not know any clinical details.

 We break from our usual habit of removing the ECG machine’s interpretation of the ECG to serve as a reminder that the computer interpretation can be wrong.  ECGs should ALWAYS be interpreted by a knowledgeable person.  The machine interpretation can serve as a reminder, but should not take the place of human interpretation. 

Here is what we DO see:  There is a normal sinus rhythm present, as evidenced by the regular P waves that do not change their morphology.  Some of the P waves are “buried” behind QRS or T waves.  The atrial rate is 95 bpm. 

The ventricular rhythm, at 40 bpm, is also regular, but is separate from the atrial rhythm.  Even though some of the P waves LOOK like they have conducted to produce QRS complexes, they have not.  The PRIs are not all the same.  Neither do they “progressively prolong”.  There is no irregularity of the QRS rhythm or variation in QRS morphology.  We see the classic “AV DISSOCIATION” of complete heart block. 

When there is a third-degree AV block with a narrow-QRS escape rhythm, we can assume the block is in the AV node.  The junction is the escape focus, producing a narrow-complex rhythm between approximately 40-60 bpm.  In this case, the QRS is slightly wide at 112 ms (.11 sec), and the QRS complexes in several leads are fragmented.  Some might argue that there is an idioventricular escape mechanism.  But, with a normal frontal plane axis, borderline width,  and no T wave inversions, the rhythm looks more supraventricular.  The R wave progresson on the precordial leads shows a persistently negative QRS with late transition in V5.  The QRS complexes in V1 and V2 appear to have pathological Q waves.  When R wave progression is not normal, we should also consider electrode misplacement.