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Wenckebach

Inferior Wall M.I. With Right Ventricular M.I.

Sat, 10/28/2017 - 18:36 -- Dawn

This ECG was recorded from a 75-year-old man with substernal chest pain and diaphoresis.  It shows a pretty classic picture of acute inferior wall M.I. The second ECG is a repeat tracing with the V4 wire moved to the V4 Right position, and it is positive for right ventricular M.I.  The patient was found to have a 100% occlusion of the right coronary artery, which was opened and stented in the cath lab.

There are several other examples of IWMI with RVMI in our archives, so we will confine this commentary to the ECG signs that make these tracings so typical of right coronary artery occlusion. Once you are familiar with the typical pattern of IWMI / RVMI, it is easy to see, even when the ST elevation is subtle (as this one certainly is NOT).

Signs of IWMI in these ECGs are

·         ST elevation in inferior leads II, III and aVF.

·         Reciprocal ST depression in leads I and aVL. 

Signs of RVMI in these ECGs are:

·         ST elevation in V4 right.

·         ST elevation in V1 without ST elevation in V2.

Acute Inferior Wall M.I. With Right Ventricular M.I. and Atrial Fibrillation

Tue, 05/19/2015 - 11:10 -- Dawn

This 31-year-old man presented to the Emergency Dept. complaining of chest pain, shortness of breath, and nausea. His heart rate on admission was 120 - 130 bpm and irregular, and the monitor showed atrial fibrillation. His rate slowed with the administration of diltiazem. His 12-lead ECG shows the classic ST elevation of inferior wall M.I. in Leads II, III, and aVF. This patient also had JVD, bibasilar rales, orthopnea, and exertional dyspnea, signs of CHF. He had no history of acute M.I., CHF, or atrial fibrillation. He offered no history of drug use or medications.

This ECG is very useful for the basic student, in that the ST elevations are readily seen, and the atrial fib is definitely irregularly-irregular. For the more advanced student, the ST depression in V2 indicates posterior wall injury, while the flat ST segment in V1 indicates a possible right ventricular M.I.  While the posterior wall is trying to depress the ST segment, the right ventricle is trying to elevate it, resulting in flattening. Also, Lead III has a greater STE than Lead II, which has been shown to be a reliable indicator of RV infarction.  This should be confirmed with a V4 right, or all chest leads done on the right side. Right ventricular injury has been shown to increase mortality, and it also requires different management of hemodynamics.

Ask The Expert

Wed, 12/05/2012 - 22:11 -- Dawn
RP - PR Reciprocity, PR Intervals

QUESTION: How do you explain the changing PR intervals in the following series of Strips, and is the mechanism related to Wenckebach conduction?

Our expert today is our very own Jason Roediger, who is an ECG Guru blogger, and frequent contributer to the Ask the Expert page.  His blog, ECG Challenges, offers you a chance to try your skills and get feedback from Jason.  He has a special interest in complex arrhythmias, and is adept at constructing laddergrams that help clarify the mechanisms involved in the rhythms. His bio can be found on the "About Us" page, or on his previous contributions to this page.

Strips courtesy of R.O. from California, USA.

ANSWER:

 

I opted to only construct a laddergram for the middle rhythm tracing rather that for all 3. The bottom strip didn't deserve one since it is only showing sinus rhythm with no ectopy. The top 2 are more-or-less identical in content. However, the middle one was of slightly better diagnostic quality.

This is a classic representation of "R-P/P-R reciprocity". There is a near-linear relationship between the P-R (or P'-R) intervals and their associated R-P (or R-P') intervals. Along the right margin of the tracing, I have compiled a list of the longest R-P interval at the top which is associated with the shortest P-R interval (i.e., 81 / 29). Conversely, at the bottom of the list, the shorted R-P interval is associated with the longest P-R (i.e., 45 / 50). The two lines that I've circled are the only inconsistancy in the logic but are well within an acceptable margin of error. 
 

 

 

 

 

This patient definitely has AV nodal disease and at a faster atrial rate, they would undoubtedly develop Type I AV block (Wenckebach periodicity) and start "dropping" beats. Whenever you encounter longer P'-R intervals on the APBs, you always have to consider the possibility that conduction jumped over from the fast pathway to the slow pathway which is the initiating mechanism seen in AVNRT. I think that all of the atrial impulses are conducting via the fast pathway (FP) here but the FP is exhibiting varying conduction delays depending on how short or long the preceding R-P interval is. As I've illustrated in the laddergram, you can see that the amount of time it takes for the sinus node to recover after an APB is essentially equal to the basic sinus cycle (e.g., 110 = 111). Dr. Charles Fisch graphically charted R-P/P-R relationships in his book "Electrocardiography of Clinical Arrhythmias" on pages 28 and 29. If you were to chart the R-P and P-R intervals on a graph, they would form a diagonal line that is about 45-degrees. If you don't have Dr. Fisch's book in your personal library, you should really buy a copy. I refer to it all the time!

 

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