This rhythm strip is recorded in two simultaneous leads, which is always preferable to one single lead.  It is a good example of atrial fibrillation with a rapid ventricular response.  Atrial fib that has not been treated will usually have a rapid ventricular rate.  This reflects the ability of the AV node to conduct a tachycardia, within limits.  The natural slow conduction of the AV node allows it to act as a "filter", preventing the huge numbers of impulses generated by the atrial fibrillation from reaching the ventricles.  In this case, about 140 beats per minute are able to make it through the AV node into the ventricles.   In some patients, preexisting cardiac conditions such as valve insufficiency or CHF may make this rate dangerous for the patient.  The rate may lower cardiac output in some people, and this must be considered in light of the fact that the loss of P waves in atrial fib also lowers cardiac output significantly.

This patient has an underlying atrial fibrillation with complete heart block and an idioventricular escape rhythm.  She was treated successfully with a permanent implanted pacemaker.

This series of strips was donated by Arnel Carmona, and was taken from a patient admitted to the hospital for a urinary tract infection.  No other history is known.   On close examination of this rhythm what do we see?

Strip 1:   Narrow-complex tachycardia with NO apparent P waves.

Strip 2:   Some irregularity, with long regular groups and still NO P waves.

Strips 3 & 4:  Grouped beating.

Strip 5:   A narrow-complex rhythm that is approximately ½ the rate of Strip 1. 

When grouped beating is seen, one should always suspect Wenckebach conduction.  Wenckebach conduction (progressively longer conduction times through the A-V conduction system) can occur in rhythyms other than sinus rhythm.  Without P waves and PR intervals, GROUPED BEATING is our major clue to Wenckebach conduction. 

This patient has an underlying atrial fibrillation – hence no P waves.  Fine fibrillatory waves can be seen, but artifact can cause the same appearance.  So, why is there no irregular irregularity?  There is another rhythm at work here along with the atrial fibrillation.  Junctional tachycardia is seen in Strip 1.  When two tachycardias coexist, one from above the AV junction, and one from below, the rhythm can be called a “double tachycardia”.  This particular combination often happens in patients with digitalis toxicity. 

In some cases, a complete heart block at the level of the atrial conduction fibers or the AV node causes  two rhythms to operate independently.  Any supraventricular rhythm, including atrial fib, can occur with a complete heart block, in which case we would see an “escape” rhythm.  Escape rhythms are usually slow, either idiojunctional (40-60 bpm) or idioventricular (< 40 bpm).  

 Let’s look at each of the strips in detail.  We will begin with the hypothesis that this is atrial fibrillation with concurrent junctional tachycardia at around 150 bpm.  I will include laddergrams to illustrate my view of what is happening. 

This is a good example of atrial fibrillation with left bundle branch block.  You get two ECGs with this one, because the patient presented to EMS with a fast heart rate, and the rate was slowed with the drug diltiazem.  We do not have any other patient information, unfortunately.

In the first strip, we see a wide-complex tachycardia.  In an emergency situation, with an unstable patient, this rhythm could safely be treated using an emergency ventricular tachycardia (VT) protocol.  In fact, all WCTs should be considered to be VT until proven otherwise.  In most emergency settings, the unstable VT patient would be electrically cardioverted, which will often convert atrial fibrillation as well.  The stable patient with this rhythm would be treated with an antiarrhythmic drug, such as amiodarone.  This may convert or slow down atrial fib.  So, in the initial stages of treatment, differentiating between VT and A Fib is not the first priority.  Assessing the patient's hemodynamic stability and addressing the rate if necessary are the priorities.  

So, how do we know this is NOT VT?  It can be difficult, but in this case, the rhythm, even though fast, is very irregular.   VT is not always perfectly regular, but this irregularly-irregular rhythm points to atrial fibrillation. Also, the pattern of the QRS morphology fits with LBBB.  The criteria for LBBB are:  1) supraventricular rhythm, 2) wide QRS, 3) negative QRS in V1 with positive QRS in Leads I and V6.  If we assume the rhythm is atrial fibrillation, we meet the first criteria.  The other two are self-evident.

After the medication is administered, 10 minutes later, we see the rate slow down.  There is no change in the irregularly-irregular rhythm, and the LBBB pattern remains.  All that has changed is the rate and, hopefully, the patient's symptoms.  This confirms that the original rhythm was not VT.  

Remember, atrial fib lowers cardiac output because there is no P wave - no "atrial kick".  Also, the fast rate associated with new-onset atrial fib often compromises ventricular filling and cardiac output.   LBBB also has a deleterious effect on cardiac output.  Wide QRS complexes indicate that the ventricles are not contracting efficiently and synchronously.  The left ventricle is depolarizing by way of a slow wave of depolarization, rather than all the cells getting the message to depolarize at the same time.  Having these two conditions at the same time can have a very negative effect on cardiac output, leading to CHF.  The first step in treatment often involves simply slowing the rate to normal, which allows for better ventricular filling and decreases the workload on the heart.  Then, the fibrillation and bundle branch block can be addressed.