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Dawn's picture

ECG Basics: Torsades Cardioverted

These two strips are from one patient who was electrically cardioverted twice in a few minutes.  The original reason for the cardioversion was Torsades de Pointes, a type of polymorphic ventricular tachycardia associated with a long QT interval.  For more information about TDP, go to this LINK.  It is a bit difficult to comment on the patient's post-cardioversion rhythm, because so little of it is shown.  It appears to be sinus, with a wide QRS.  The QT interval appears slightly prolonged at .44 sec, but it is not known what the QT interval is corrected to a rate of 60/min.  TDP is often seen with QT intervals greater than 600 ms (.6 seconds).  Also THESE STRIPS ARE NOT SIMULTANEOUS, they were taken two minutes apart.  In the first one, the P waves and T waves look so much alike, they could all be P waves.  They do not "march out".  It is necessary to get a long strip, preferably in multiple leads, and a 12-Lead ECG, to properly evaluate the rhythm post-cardioversion. 

Dawn's picture

Teaching Series 1113: ECG 5 of 6 - Acute Anterior Wall M.I.

Continuing our series from the patient with acute AWMI, donated by Jenda Enis Štros, a new dysrhythmia has appeared.  The patient was taken to the cath lab, and a thrombus was removed from the stent.   We now see a wide-complex tachycardia.  The morphology (shape and configuration) of the QRS has changed considerably from the previous ECGs, so we know this is idioventricular in origin.  There are many other clues, but  some major ones are:  the QRS is very wide, there is no associated P wave, all the precordial leads except V1 are negative (precordial concordance), V6 is negative, and the axis is away from II, III, and aVF and toward aVR (aVR is upright).  

Because the rate is about 125/min., this could be called ventricular tachycardia (V Tach) or accelerated idioventricular rhythm (AIVR).  The important fact here is that this rhythm was transitory and the patient remained stable.  Reperfusion dysrhythmias are not uncommon, and the patient is treated as indicated by the clinical condition. 

Here are links to all six ECGs in this series:

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-1-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-2-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-3-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-4-6-acute-anterior-wall-mi

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-5-6-acute-anterior-wall-mi 

http://www.ecgguru.com/ecg/teaching-series-1113-ecg-6-6-acute-anterior-wall-mi

 

   

Dawn's picture

Teaching Tip: 12 Leads are Better Than One (Or Three)

Years ago, I was tasked with introducing 12-lead ECG interpretation to firefighter/paramedics who had been using ECG for rhythm monitoring for years.  Some were eager to add to their skills, others - not so much.  The feeling was, we have been doing just fine as we are.  When finally convinced that they could interpret STEMI with a 12-lead, many were content to use the 12-lead ECG only for that.  

To illustrate to students the great value of multi-lead assessment, I devised a little "quiz".  I showed the students ten to twelve short rhythm strips, like you see here.  All were cropped from 12-lead ECGs.  I asked my class to interpret the strips as they would if they were taking an ACLS class.  Usually, all did fine, or so they thought.  When shown the 12-lead ECGs the strips were taken from, EVERY student changed his or her mind on EVERY ECG.  The lesson is:  sometimes what we are looking for shows up in some leads and not others.  You can find this illustrated hundreds of times just in the ECG archives on this site.  I will supply some ECGs here on this page over the next few weeks that you could use to show your own students the value of "multi-lead assessment".  

What started as a hard-sell turned out to be a fun exercise.

The ECG shown here is of a patient in V Tach.  There are several strong signs that this is V Tach, including the wide QRS complexes, lack of associated P waves, "backward" axis, also called extreme right axis deviation (Leads II, III, and aVF are all negative and aVR is positive), and V6 is negative.  For more review of the differential diagnosis of wide-complex tachycardias, go to our Ask the Expert answer from Jason Roediger.  This LINK willl take you to Dr. Grauer's informative webpage where he offers a step-by-step guide to differentiating the WCTs.

 The focus of THIS lesson is that, while the patient is in V Tach, and it is in every lead, the tell-tale signs are harder to see in some leads than others.  Remember to show your  Remember to share with your students that the channels of the ECG (in this case three) are run simultaneously, so that the same heartbeat is seen several times - once for each channel.

Dawn's picture

ECG Basics: Ventricular Tachycardia

An example of ventricular tachycardia in Lead II.  This patient's rate is about 190/min.  V Tach will have the following criteria:  Rate greater than 100/min, QRS duration greater than .12 sec. (120 ms), and no P wave associated with the QRS.

It can be difficult to distinguish V Tach from other wide-complex tachycardias without a 12-lead ECG, but all wide-complex tachycardias should be treated as V Tach until proven otherwise, as V Tach is a potentially lethal dysrhythmia.  V Tach can cause a severe reduction in cardiac output which can lead to V Fib and death.

Dawn's picture

ECG Basics: Torsades de Pointes

Torsades de pointes, or polymorphic ventricular tachycardia, is a ventricular tachycardia precipitated by and associated with long QT Syndrome.  Long QT Syndrome can be congenital or acquired.  Torsades is life-threatening, and can be made worse by many drugs, including some of the drugs used to treat VT.  The rate is usually 150 - 250 / min. and the appearance is of a wide-complex tachycardia with QRS morphology changes.  In some leads, it will appear as if it is "twisting" around the isoelectric line, giving it the French name, Torsades de pointes, a ballet term meaning twisting of the points.  For a thorough discussion of Torsades, check this LINK.

Dawn's picture

Ventricular Tachycardia

This is a good example of ventricular tachycardia with PRECORDIAL CONCORDANCE.  The QRS complexes in the chest, or precordial, leads all point downward.  When the precordial leads are all  negative or all positive in a wide-complex tachycardia, there is virtually a 100% chance that the WCT is ventricular tachycardia.  This ECG shows many characteristics of VT, including the extreme "backwards" axis:  aVR is positive and II, III, and aVF are negative.  Lead I is almost equiphasic.  Also, the lack of a clear BBB pattern and a negative V6 are strongly suggestive of VT.  REMEMBER:  In the treatment of wide-complex tachycardia, the rhythm should be considered VT unless proven otherwise.  This is especially true in unstable patients, patients over 50 years old, and patients with known heart disease.

INSTRUCTORS' NOTE:  We purposely left the machine interpretation on this week's ECG of the Week.  How many errors did the machine make?  This might be a good teaching point for students of all levels.

 

jer5150's picture

Jason's Blog: ECG Challenge of the Bi-Week for Nov. 18th - Dec. 1st.

Patient's clinical data:  64-year-old white man.

What is the rhythm seen in this 12-lead ECG?

Dawn's picture

Ventricular Tachycardia

A 66 year old man is complaining of palpitations and chest pain which radiated to his left arm and neck, which lasted 20 minutes, then went away.  Paramedics found him in V Tach.  His BP was 120/80 and his pulse 120/min. He converted to a narrow-complex rhythm while being given amiodarone, but became nauseated.  He returned to V Tach, and his symptoms disappeared.  This patient had an implanted defibrillator, which never went off.  How do we know this is V Tach?

First, ALWAYS consider any wide-complex tachycardia to be VT unless you have proof that it is not.  When symptoms include chest pain, it can be especially dangerous to miss the diagnosis of VT.  Remember, some VT can be asymtomatic, even for prolonged periods of time.

ECG signs that this is VT include: QRS is extremely wide (>.14 sec), no P waves associated with the QRS complexes, negative complexes in V4, V5 and V6.  In fact, this patient has negative "precordial concordance" - all the chest leads are negative. This is a strong sign of VT.  The wide little r wave in V1, greater than .04 sec (one small block) is a strong indicator of VT, as is the delayed nadir of the S waves in V1 through V3 (the slope of the S wave is not steep, indicating a long time to depolarize the ventricles).

For more about V Tach criteria, browse this site and visit the ECG blogs on the Favorites page.  It is a favorite topic among ECG experts.

 

Dawn's picture

Wide-Complex Tachycardia Converted by Adenosine

This ECG was presented earlier this week as an example of SVT with LBBB aberrancy, which was ultimately converted with one dose of adenosine in the Emergency Department.  It is the most shared and commented on ECG yet to appear on the Guru.  The diagnosis given was the one accepted by the medical staff who cared for the patient, who was a man in his 30's who presented to the Emergency Department complaining of a rapid heart rate.  He was ambulatory with stable vital signs, in spite of the tachycardia. He reported that he has had several episodes of fast heart rate which responded to either Valsalva maneuvers or, in some cases, medication in the ED.  He was told he might benefit from an ablation procedure, but he did not have health insurance and continued to use the ED as his primary source of medical care.  When he was admitted to the ED, the tech initially called for help, thinking the monitor showed ventricular tachycardia.  The ED physician felt that this represented LBBB aberrancy, possibly rate-dependent, and he treated the patient with adenosine.  The rhythm converted to sinus after one dose, and the patient remained stable throughout the process.  He was advised to undergo further observation and testing, but he declined due to financial concerns, and the fact that he usually succeeded in relieving his symptoms with "bearing down".  

Wide-complex tachycardias can be difficult to assess simply from an ECG. The patient's stability depends more upon general health and cardiac output issues than the origin of the tachycardia.  When we presented this ECG, we also presented the diagnosis he had upon discharge from the ED.

Subsequently, ECG Guru Dr. Ken Grauer, a frequent contributer to this site, offered his alternative diagnosis and his explanation of why he believes this to be v tach.  Other well-respected ECG experts have also questioned the original diagnosis.  Please refer to the comments below for this very helpful explanation.  Unfortunately, this patient is lost to followup, as this incident occurred some time ago.

WTCs remain a most fascinating topic, especially for those who enjoy "detective work".  We thank Dr. Grauer, Tom Bouthillet, and others for their contributions to the ECG Guru on this topic.
   

Dawn's picture

AV Sequential Pacing to Ventricular Tachycardia

This is an interesting ECG for showing students AV sequential pacing and also ventricular tachycardia.  The unusual thing about this ECG is that the V Tach starts at the time the machine begins recording the precordial leads.  This particular ECG machine shows a slight "gap" at the lead change, so we don't see the actual start of the V Tach.  Both rhythms have wide QRS complexes. The pacemaker is pacing the right ventricle, so you will see a wide QRS with a leftward axis, as the impulse spreads up and leftward toward the left ventricle.   The V Tach portion is, of course, limited to the precordial leads, so we cannot plot the frontal plane axis.  But, it meets many of the accepted criteria for ventricular tachycardia, including:  very wide QRS, negative QRS in Lead V6, absence of RBBB or LBBB pattern.   For more on recognizing V Tach in a WCT, go to Ask the Expert at this LINK.    

This is also a very good example of how the interpretation by the machine can be wrong.  Always read the ECG yourself!

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